Search results for "Right ventricular"

showing 10 items of 44 documents

Molecular disturbance underlies to arrhythmogenic cardiomyopathy induced by transgene content, age and exercise in a truncated PKP2 mouse model

2016

13 páginas, 9 tablas, 2 figuras. Contiene material suplementario.

0301 basic medicineGenetically modified mouseAgingmedicine.medical_specialtyTransgeneCardiomyopathyPlakoglobinConnexin030204 cardiovascular system & hematologyBiologyMice03 medical and health sciences0302 clinical medicineFibrosisInternal medicineGeneticsmedicineAnimalsHumansTransgenesMolecular BiologyArrhythmogenic Right Ventricular DysplasiaGenetics (clinical)General Medicinemedicine.diseasePhenotypeDisease Models Animal030104 developmental biologyEndocrinologyMutationDisease ProgressionPhysical EnduranceDesminPlakophilins
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The EP300/TP53 pathway, a suppressor of the Hippo and canonical WNT pathways, is activated in human hearts with arrhythmogenic cardiomyopathy in the …

2021

Aim Arrhythmogenic cardiomyopathy (ACM) is a primary myocardial disease that typically manifests with cardiac arrhythmias, progressive heart failure and sudden cardiac death (SCD). ACM is mainly caused by mutations in genes encoding desmosome proteins. Desmosomes are cell-cell adhesion structures and hubs for mechanosensing and mechanotransduction. The objective was to identify the dysregulated molecular and biological pathways in human ACM in the absence of overt heart failure. Methods and results Transcriptomes in the right ventricular endomyocardial biopsy samples from three independent individuals carrying truncating mutations in the DSP gene and 5 control samples were analyzed by RNA-S…

0301 basic medicinePhysiologyCardiomyopathy030204 cardiovascular system & hematologyBiologyMechanotransduction CellularBiological pathway03 medical and health sciences0302 clinical medicinePhysiology (medical)medicineHumansMechanotransductionEP300Wnt Signaling PathwayArrhythmogenic Right Ventricular DysplasiaHeart FailureHippo signaling pathwayWnt signaling pathwayArrhythmias CardiacOriginal Articlesmedicine.diseaseCell biologyDeath Sudden Cardiac030104 developmental biologyCardiomyopathy Gene expression Hippo pathway RNA-Sequencing TP53 WNT pathwayHeart failureTumor Suppressor Protein p53Signal transductionCardiomyopathiesCardiology and Cardiovascular MedicineE1A-Associated p300 ProteinCardiovascular Research
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JAK2 mediates lung fibrosis, pulmonary vascular remodelling and hypertension in idiopathic pulmonary fibrosis: an experimental study

2018

BackgroundPulmonary hypertension (PH) is a common disorder in patients with idiopathic pulmonary fibrosis (IPF) and portends a poor prognosis. Recent studies using vasodilators approved for PH have failed in improving IPF mainly due to ventilation (V)/perfusion (Q) mismatching and oxygen desaturation. Janus kinase type 2 (JAK2) is a non-receptor tyrosine kinase activated by a broad spectrum of profibrotic and vasoactive mediators, but its role in PH associated to PH is unknown.ObjectiveThe study of JAK2 as potential target to treat PH in IPF.Methods and resultsJAK2 expression was increased in pulmonary arteries (PAs) from IPF (n=10; 1.93-fold; P=0.0011) and IPF+PH (n=9; 2.65-fold; P<0.00…

0301 basic medicinePulmonary and Respiratory Medicinemedicine.medical_specialtyHypertension PulmonaryBlotting WesternMyocytes Smooth MuscleFluorescent Antibody TechniqueVasodilationVascular RemodelingReal-Time Polymerase Chain ReactionVascular remodelling in the embryo03 medical and health sciencesIdiopathic pulmonary fibrosisTransforming Growth Factor betaRight ventricular hypertrophyInternal medicinepulmonary hypertensionAnimalsHumansMedicineRNA Small InterferingRats WistarCells CulturedCell ProliferationBKCaJanus kinase 2biologybusiness.industryEndothelial CellsJanus Kinase 2idiopathic pulmonary fibrosismedicine.diseaseImmunohistochemistryPulmonary hypertensionIdiopathic Pulmonary FibrosisTriterpenesRatsPhenotype030104 developmental biologyJAK2biology.proteinCardiologyAnimal studiesJanus kinasebusinessSignal TransductionPulmonary artery smooth muscle cells Pulmonary artery endothelial cells.Thorax
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Anatomical features and clinical correlations in Caucasian patients with definite arrhythmogenic right ventricular dysplasia/cardiomyopathy.

2014

AIM: Arrhythmogenic right ventrticular dysplasia/cardiomyopathy (ARVD/C) is an inherited cardiomyopathy characterized by fibrofatty replacement and a high risk of ventricular arrhythmias (VA) and sudden cardiac death (SCD). The aim of the present investigation is to examine the pathological profile and the clinical correlations in a group of ARVD/C patients. METHODS: We conducted a multicenter study evaluating 47 patients (31 men; mean age 37±14 years) with definite ARVD/C. Diagnosis was established according to the actual clinicomorphologic criteria at autopsy or clinically. We divided the study population in 2 different groups. First group included 28 alive patients and the second 19 pati…

AdultMaleAdolescentEuropean Continental Ancestry GroupLeftAge FactorsAdolescent; Adult; Age Factors; Aged; Arrhythmogenic Right Ventricular Dysplasia; Death Sudden Cardiac; European Continental Ancestry Group; Humans; Male; Middle Aged; Retrospective Studies; Ventricular Dysfunction Left; Young AdultMiddle AgedSettore MED/11 - Malattie Dell'Apparato CardiovascolareSuddenWhite PeopleDeathVentricular Dysfunction LeftYoung AdultDeath Sudden CardiacVentricular DysfunctionHumansCardiomyopathies - Death sudden - Young adult - ExerciseCardiacArrhythmogenic Right Ventricular DysplasiaAgedRetrospective StudiesMinerva cardioangiologica
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Clinical characteristics and determinants of the phenotype in TMEM43 arrhythmogenic right ventricular cardiomyopathy type 5.

2020

Arrhythmogenic right ventricular cardiomyopathy type V (ARVC-5) is the most aggressive heterozygous form of ARVC. It is predominantly caused by a fully penetrant mutation (p.S358L) in the nondesmosomal gene TMEM43-endemic to Newfoundland, Canada. To date, all familial cases reported worldwide share a common ancestral haplotype. It is unknown whether the p.S358L mutation by itself causes ARVC-5 or whether the disease is influenced by genetic or environmental factors. The purpose of this study was to examine the phenotype, clinical course, and the impact of exercise on patients with p.S358L ARVC-5 without the Newfoundland genetic background. We studied 62 affected individuals and 73 noncarrie…

AdultMalemedicine.medical_specialtyDNA Mutational AnalysisMutation MissenseDisease030204 cardiovascular system & hematologyVentricular Function LeftRight ventricular cardiomyopathySudden cardiac deathElectrocardiography03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicinemedicineGeneticsHumans030212 general & internal medicineExerciseArrhythmogenic Right Ventricular DysplasiaEjection fractionTMEM43business.industryIncidence (epidemiology)HaplotypeMembrane ProteinsStroke VolumeDNAmedicine.diseasePhenotypePedigree3. Good healthPhenotypeMutation (genetic algorithm)CardiologyFemaleCardiology and Cardiovascular MedicinebusinessArrhythmogenic right ventricular cardiomyopathyArrhythmia
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Left ventricular Myocardial dysfunction in arrhythmogenic cardiomyopathy with left ventricular involvement: A door to improving diagnosis.

2018

[EN] Background: Diagnostic Task Force Criteria (TFC) for arrhythmogenic cardiomyopathy (AC) exhibit poor performance for left dominant forms. TFC only include right ventricular (RV) dysfunction (akinesia, dyssynchrony, volumes and ejection fraction). Moreover, cardiac magnetic resonance imaging (CMRI) assessment of left ventricular (LV) dyssynchrony has hitherto not been described. Thus, we aimed to comprehensively characterize LVCMRI behavior in AC patients. Methods: Thirty-five AC patients with LV involvement and twenty-three non-affected family members (controls) were enrolled. Feature-tracking analysis was applied to cine CMRI to assess LV ejection fraction (LVEF), LV end-systolic and …

AdultMalemedicine.medical_specialtyHeart VentriclesCardiomyopathyMagnetic Resonance Imaging Cine030204 cardiovascular system & hematologyVentricular Function LeftStrainTECNOLOGIA ELECTRONICA03 medical and health sciencesVentricular Dysfunction Left0302 clinical medicineCardiac magnetic resonance imagingDiastoleInternal medicinemedicineLate gadolinium enhancementHumansCor030212 general & internal medicineLeft ventricular involvementVentricular dysfunctionCardiac magnetic resonance imagingArrhythmogenic Right Ventricular DysplasiaEjection fractionmedicine.diagnostic_testTask forcebusiness.industryLeft ventricular arrhythmogenic cardiomyopathyReproducibility of ResultsStroke VolumeMiddle Agedmedicine.diseasePatologiaDyssynchronyLv dyssynchronyCardiologyFemaleCardiology and Cardiovascular MedicineLEFT DOMINANTbusiness
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Therapeutic management in Sicilian patients with definite arrhythmogenic right ventricular dysplasia/cardiomyopathy and focus on the role of implanta…

2014

Arrhythmogenic Right Ventrticular Dysplasia/Cardiomyopathy(ARVD/C) is an inherited cardiomyopathy characterized by right ven-tricularmyocytelosswithfibrofattyreplacement,ahighriskofventric-ular arrhythmias (VA) and sudden cardiac death (SCD) [1]. Preventionof SCD represents the most important management strategy and theachievement of this target can be reached by different therapeuticstrategies including implantable cardioverter-defibrillator (ICD)implantation, pharmacologic therapy, catheter ablation of ventriculartachycardia (VT) and cardiac transplantation [2,3]. The aim of thisstudy is to examine the outcome of the different therapies adopted ina group of affected patients, focusing on th…

AdultMalemedicine.medical_specialtyTime FactorsCardiomyopathymedicine.medical_treatmentCardiomyopathyCatheter ablationImplantable cardioverterdefibrillatorSudden deathInternal medicinemedicineHumansMedical managementSicilySurvival rateArrhythmogenic Right Ventricular DysplasiaCardiomyopathy; Implantable cardioverterdefibrillator; Medical management; Predictors; Sudden deathSudden deathbusiness.industryPrognosisImplantable cardioverter-defibrillatormedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareDefibrillators ImplantableArrhythmogenic right ventricular dysplasiaSurvival RateTransplantationDeath Sudden CardiacTreatment OutcomeHeart failureCardiologyFemaleMorbidityCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesPredictorInternational Journal of Cardiology
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An unusual case of sudden cardiac death during sexual intercourse

2018

The most common cause of sudden death during sexual intercourse in adults is heart disease, and it is usually the male, whereas the death of the woman is unusual. Generally, in these cases, death occurs as a result of cardiovascular disease. The authors report an unusual case of the sudden death of a young woman during sexual intercourse. The post-mortem investigations (autopsy, cardiac nuclear magnetic resonance and cardiac histology) demonstrated a previously undiagnosed arrhythmogenic right ventricular cardiomyopathy. The terminal cause of death was a malignant arrhythmia from arrhythmogenic right ventricular cardiomyopathy. This is the first report of a case in which sexual activity ca…

Adultmedicine.medical_specialtyHeart diseaseundiagnosed heart diseaseAutopsyDeath of a young woman during sexual intercourse; analysis of factors leading to death; autopsy; stress of normal intercourse; suspicion of murder; undiagnosed heart diseaseDisease030204 cardiovascular system & hematologySudden deathDeath of a young woman during sexual intercourseRight ventricular cardiomyopathySudden cardiac death03 medical and health sciencesanalysis of factors leading to death0302 clinical medicineSettore MED/43 - Medicina LegaleInternal medicinemedicineHumans030216 legal & forensic medicineArrhythmogenic Right Ventricular DysplasiaCause of deathbusiness.industrystress of normal intercourseGeneral Medicinemedicine.diseaseMagnetic Resonance ImagingSexual intercourseDeath Sudden Cardiacsuspicion of murdercardiovascular systemCardiologyFemaleSexAutopsybusinessMedico-Legal Journal
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Assessment of the Tricuspid Valve Morphology by Transthoracic Real-Time-3D-Echocardiography

2005

Aim: To demonstrate the feasibility of transthoracic three-dimensional real-time echocardiography (3D-TTE) supplemental to routine assessments of the tricuspid valve and to analyze interrater agreement. Methods: Twenty healthy subjects and 74 patients with right ventricular failure were examined with conventional 2D and additionally 3D-TTE (SONOS 7500, Philips, Netherlands). The 3D exams were performed and recorded by one of two raters. The recordings were evaluated offline and independently by both raters for visualization of morphological and functional features of the tricuspid valve according to a subjective 3-point scale. Statistical analyses were performed for interrater agreement and…

Adultmedicine.medical_specialtyTime FactorsFunctional featuresEchocardiography Three-DimensionalReal time 3d echocardiographyInternal medicinemedicineHumansRadiology Nuclear Medicine and imagingProspective cohort studyHeart FailureObserver VariationTricuspid valvebusiness.industryHealthy subjectsTricuspid Valve InsufficiencyInter-rater reliabilitymedicine.anatomical_structureImaging qualitycardiovascular systemCardiologyFeasibility StudiesRight ventricular failureFemaleTricuspid ValveRadiologyCardiology and Cardiovascular MedicinebusinessEchocardiography
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Incidence and Clinical Impact of Right Ventricular Involvement (Biventricular Ballooning) in Takotsubo Syndrome: Results From the GEIST Registry

2021

Background: The short- and long-term prognosis of Takotsubo syndrome (TTS) presenting with right ventricular (RV) involvement remains poorly understood. Research Question: What is the incidence and clinical outcome of RV involvement in TTS? Study Design and Methods: This study analyzed 839 consecutive patients with TTS (758 female subjects and 81 male subjects) in a multicenter registry. RV involvement was defined as wall motion abnormality of the RV free wall, with or without apical involvement. The median long-term follow-up was 2.1 years (interquartile range, 0.3-4.5 years). The primary outcome was in-hospital and out-of-hospital all-cause mortality. The secondary end point was a composi…

Aged 80 and overMaleIncidenceVentricular Dysfunction Rightshort- and long-term outcomeShock CardiogenicArrhythmias CardiacPulmonary EdemaMiddle AgedPrognosisVentricular Dysfunction Leftright ventricular involvementTakotsubo CardiomyopathyThromboembolismHumansFemaleHospital MortalityRegistriesMortalityTakotsubo syndromeAged
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